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By Lee L. Swanstrom, Christy M. Dunst

This quantity offers a accomplished, cutting-edge review of the most important concerns particular to the sector of antireflux surgical procedure. It presents remarkable tutorial aspect relating to functionality of antireflux surgical procedure from major esophageal surgeons all over the world. This quantity represents the one source of its variety devoted particularly to the problems certain to antireflux surgical procedure. it's wealthy intimately and precious illustrations that train surgeons in right approach in addition to proposing the reasoning at the back of a number of recommendations. Written by way of specialists within the box, Antireflux surgical procedure is of significant price to practising surgeons who practice gastrointestinal surgical procedure, scientific scholars, surgical citizens, and fellows.

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Morphology of the cardia and significance of carditis in pediatric patients. Am J Surg Pathol. 2002;26:1032–9. 55. Paull A, Trier JS, Dalton MD, Camp RC, Loeb P, Goyal RK. The histologic spectrum of Barrett’s esophagus. N Engl J Med. 1976; 295:476–80. 56. Spechler SJ, Zeroogian JM, Antonioli DA, Wang HH, Goyal RK. Prevalence of metaplasia at the gastroesophageal junction. Lancet. 1994;344:1533–6. 57. Chandrasoma PT, Der R, Dalton P, Kobayashi G, Ma Y, Peters J, DeMeester T. Distribution and significance of epithelial types in columnar lined esophagus.

The distal 3–4 cm of the segment in Paull et al. consisted of cardiac and oxyntocardiac epithelia [55]. The goal of treating reflux with acid suppressive drugs is to alkalinize gastric juice; the unintended consequence of this is that the esophagus is exposed to alkalinized material during reflux. This results in increased prevalence and extent of intestinal metaplasia in the columnar lined segment. Carcinogenesis in Columnar Lined Esophagus The only epithelium in the esophagus that is susceptible to carcinogenesis is intestinal epithelium [51].

In: Chandrasoma PT, DeMeester TR, editors. GERD: reflux to esophageal adenocarcinoma. San Diego: Academic; 2006. p. 89–106. 46. Chandrasoma PT, DeMeester TR. Chapter 9: the pathology of reflux disease at a cellular level: part 2 – evolution of cardiac mucosa to oxyntocardiac mucosa and intestinal metaplasia. In: Chandrasoma PT, DeMeester TR, editors. GERD: reflux to esophageal adenocarcinoma. San Diego: Academic; 2006. p. 169–200. 26 47. Yamanaka Y, Shiotani A, Fujimura Y, Ishii M, Fujita M, Matsumoto H, Tarumi K, Kamada T, Hata J, Haruma K.

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